Now showing items 1-5 of 5

    • Fuentes, Marisol; Hernández, Romané; Gordillo, Diego; Amaro, José; Falconer, Mary A.; Alburquenque, Claudio; Tapia, Cecilia (Sociedad Chilena de Infectologia, 2014)
      Background: Melanocytes are cells located in epidermis and mucous membranes that synthesize melanin and cytokines. It is known that melanin has antimicrobial activity and that melanocytes are melanized in presence of ...
    • Fuentes, Marisol; Hermosilla, Germán; Alburquenque, Claudio; Falconer, Mary A.; Amaro, José; Tapia, Cecilia (Sociedad Chilena de Infectologia, 2014)
      © 2014 Sociedad Chilena de Infectologia. All rights reserved. Abstract Introduction: The commensal yeast Candida albicans, can cause superficial or systemic candidiasis in susceptible hosts. In Chile, azole antifungals are ...
    • Tapia Paredes, Cecilia; Hermosilla Díaz, Germán; Fortes, Paula; Alburquenque, Claudio; Bucarey, Sergio; Salinas, Hugo; Rodas, Paula I.; Díaz Jarabrán, María Cristina; Magne, Fabien (Springer Netherlands, 2017)
      To study Candida albicans genotypes using RAPD and their susceptibility to fluconazole in healthy pregnant women and in vulvovaginal candidiasis (VVC) patients after topical treatment with clotrimazole. Methods: Vaginal ...
    • Tapia, Cecilia; Falconer, Maryanne; Tempio, Fabián; Falcón, Felipe; López, Mercedes; Fuentes, Marisol; Alburquenque, Claudio; Amaro, José; Bucarey, Sergio A.; Nardo, Anna Di (Oxford University Press, 2014)
      Melanocytes are dendritic cells located in the skin and mucosae that synthesize melanin. Some infections induce hypo-or hyperpigmentation, which is associated with the activation of Toll-like receptors (TLRs), especially ...
    • Alburquenque, Claudio; Amaro, José; Fuentes, Marisol; Falconer, Mary A.; Moreno, Claudia; Covarrubias, Cristian; Pinto, Cristian; Rodas, Paula I.; Bucarey, Sergio A.; Hermosilla, Germán; Magne, Fabien; Tapia, Cecilia V. (Oxford University Press, 2019)
      Candida albicans is commensal yeast that colonizes skin and mucosa; however, it can become an opportunist pathogen by changing from blastoconidia (commensal form) into hypha (pathogenic form). Each form activates a different ...