Now showing items 1-3 of 3

    • Cea Pisani, Luis; Balboa, Elisa; Puebla, Carlos; Vargas, Aníbal; Cisterna, Bruno; Escamilla, Rosalba; Regueira, Tomás; Sáez, Juan C. (Elsevier, 2016)
      Long-term treatment with high glucocorticoid doses induces skeletal muscle atrophy. However, the molecular mechanism of such atrophy remains unclear. We evaluated the possible involvement of connexin-based hemichannels (Cx ...
    • Cea Pisani, Luis Andrés; Puebla, Carlos; Cisterna, Bruno A.; Escamilla, Rosalba; Vargas, Anibal A.; Frank, Marina; Martinez Montero, Paloma; Prior, Carmen; Molano, Jesus; Esteban Rodríguez, Isabel; Pascual, Ignacio; Gallano, Pía; Lorenzo, Gustavo; Pian, Héctor; Barrio, Luis C.; Willecke, Kla; Saez, Juan C. (Springer, 2016)
      Skeletal muscles of patients with Duchenne muscular dystrophy (DMD) show numerous alterations including inflammation, apoptosis, and necrosis of myofibers. However, the molecular mechanism that explains these changes remains ...
    • Balboa, Elisa; Saavedra Leiva, Fujiko; Cea Pisani, Luis Andrés; Vargas, Anibal A.; Ramírez, Valeria; Escamilla, Rosalba; Saez, Juan C.; Regueira, Tomás (Lippincott Williams & Wilkins, 2018)
      Skeletal muscles (similar to 50% of the body weight) are affected during acute and late sepsis and represent one sepsis associate organ dysfunction. Cell membrane changes have been proposed to result from a channelopathy ...