Browsing by Author "7e59b41a-2461-408e-b5ad-765082995ae8"

Now showing items 1-4 of 4

    • Activation of the NLRP3 inflammasome in dendritic cells induces IL-1Β-dependent adaptive immunity against tumors 
      Ghiringhelli, François; Apetoh, Lionel; Tesniere, Antoine; Aymeric, Laetitia; Ma, Yuting; Ortiz, Carla; Vermaelen, Karim; Panaretakis, Theocharis; Mignot, Grégoire; Ullrich, Evelyn; Perfettini, Jean Luc; Schlemmer, Frédéric; Tasdemir, Ezgi; Uhl, Martin; Génin, Pierre; León C., Paulo C. (Nature Publishing Group, 2009)
      The therapeutic efficacy of anticancer chemotherapies may depend on dendritic cells (DCs), which present antigens from dying cancer cells to prime tumor-specific interferon-γ (IFN-γ)-producing T lymphocytes. Here we show ...
    • Erratum: Mitochondrial control of cell death induced by hyperosmotic stress (Apoptosis (2007) 12:3 DOI: ... 
      Criollo Céspedes, Alfredo; Galluzzi, Lorenzo; Chiara Maiuri, M.; Tasdemir, Ezgi; Lavandero González, Sergio; Kroemer, Guido (2013)
    • IKK connects autophagy to major stress pathways 
      Criollo Céspedes, Alfredo; Senovilla, Laura; Authier, Hélène; Maiuri, Maria Chiara; Morselli, Eugenia; Vitale, Ilio; Kepp, Oliver; Tasdemir, Ezgi; Galluzzi, Lorenzo; Shen, Si; Tailler, Maximilien; Delahaye, Nicolas; Tesniere, Antoine; De Stefano, Daniela; Younes, Aména Ben; Harp (Taylor and Francis Inc., 2010)
      Cells respond to stress by activating cytoplasmic mechanisms as well as transcriptional programs that can lead to adaptation or death. Autophagy represents an important cytoprotective response that is regulated by both ...
    • Senescence, apoptosis or autophagy? When a damaged cell must decide its path - A mini-review 
      Vicencio Bustamante, José Miguel; Galluzzi, Lorenzo; Tajeddine, Nicolas; Ortiz, Carla; Criollo Céspedes, Alfredo; Tasdemir, Ezgi; Morselli, Eugenia; Ben Younes, Amena; Maiuri, Maria Chiara; Lavandero González, Sergio; Kroemer, Guido (2008)
      Many features of aging result from the incapacity of cells to adapt to stress conditions. When damage accumulates irreversibly, mitotic cells from renewable tissues rely on either of two mechanisms to avoid replication. ...