Browsing by Author "XX676264"
Now showing items 1-6 of 6
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Zamorano, Francisco; Billeke, Pablo; Kausel, Leonie; Larrain, Josefina; Stecher, Ximena; Hurtado, José M.; López, Vladimir; Carrasco Chaparro, Ximena; Aboitiz, Francisco (Nature Publishing Group, 2017)Attention Deficit Hyperactivity Disorder (ADHD) is the most common neuropsychiatric disorder in childhood and is characterized by a delay of cortical maturation in frontal regions. In order to investigate interference ...
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López, Vladimir; Ortega, Rodrigo; Moenne, Cristóbal; Carrasco Chaparro, Ximena (Elsevier Science BV., 2016)Mu rhythm is an EEG oscillatory measure (9 -13 Hz), whose suppression during movement execution or action observation is considered an indicator of the activity of the human analog of the mirror neuron system. Detection ...
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Ortega Jiménez, Nelson Rodrigo; López, Vladimir; Carrasco Chaparro, Ximena; Escobar, María Josefina; García, Adolfo M.; Parra, Mario A.; Aboitiz, Francisco (Nature, 2020)Working memory (WM) impairments in ADHD have been consistently reported along with deficits in attentional control. Yet, it is not clear which specific WM processes are affected in this condition. A deficient coupling ...
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Navarro Cruzat, Johan Cristopher; Núñez Santander, Catalina Valeria (Universidad de Chile, 2012)La Dispraxia en nuestro país es un tema poco investigado, subdiagnosticado y frecuentemente considerado sólo como un problema menor, pese a que trae considerables consecuencias en la vida del niño que la padece. Según ...
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Wainstein, G.; Rojas Libano, D.; Crossley, N. A.; Carrasco Chaparro, Ximena; Aboitiz, F.; Ossandon, T. (Nature Publishing Group, 2017)Attention-deficit/hyperactivity disorder (ADHD) diagnosis is based on reported symptoms, which carries the potential risk of over- or under-diagnosis. A biological marker that helps to objectively define the disorder, ...
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Narváez, Carolina; Lacaux Uribe, Patricio; Cortés, Camila; Manterola Mordojovich, Carla; Carrasco Chaparro, Ximena (Sociedad Chilena de Pediatría, 2020)La deficiencia del transportador de glucosa tipo 1 constituye un síndrome (SD-GLUT1), provocado por la mutación del gen SLC2A1, que codifica la proteína transportadora de glucosa al encéfalo. Las manifestaciones neurológicas ...