Browsing by Author "fdec72c9-71ad-4f64-8034-b9d7f60389f2"

Now showing items 1-5 of 5

    • Neuroinflammation in the pathogenesis of Alzheimer's disease. A rational framework for the search of novel ... 
      Morales, Inelia; Guzmán Martínez, Leonardo; Cerda Troncoso, Cristóbal; Farías, Gonzalo A.; Maccioni Baraona, Ricardo (Frontiers Research Foundation, 2014)
      Alzheimer disease (AD) is the most common cause of dementia in people over 60 years old. The molecular and cellular alterations that trigger this disease are still diffuse, one of the reasons for the delay in finding an ...
    • Tau oligomers and fibrils induce activation of microglial cells 
      Morales, Inelia; Jiménez, José M.; Mancilla, Marcela; Maccioni Baraona, Ricardo (IOS Press, 2013)
      Neuroinflammation is a process related to the onset of several neurodegenerative disorders, including Alzheimer's disease (AD). Increasing sets of evidence support the major role of deregulation of the interaction patterns ...
    • The natural product curcumin as a potential coadjuvant in Alzheimer’s treatment 
      Morales, Inelia; Cerda Troncoso, Cristóbal; Andrade, Víctor; Maccioni Baraona, Ricardo (IOS Press, 2017)
      Alzheimer's disease (AD) is a neurodegenerative disease characterized by a progressive cognitive impairment of patients, affecting around 12% of people older than 65 years old. WHO estimated that over 48.6 million all over ...
    • The Revitalized Tau Hypothesis on Alzheimer's Disease 
      Maccioni Baraona, Ricardo; Farías, Gonzalo; Morales, Inelia; Navarrete, Leonardo (2010)
      Many hypotheses have been raised regarding the pathophysiology of Alzheimer's disease (AD). Because amyloid beta peptide (Aβ) deposition in senile plaques appears as a late, nonspecific event, recent evidence points to tau ...
    • The Revitalized Tau Hypothesison Alzheimer's Disease 
      Maccioni Baraona, Ricardo; Farías, Gonzalo; Morales, Inelia; Navarrete, Leonardo (Elsevier Inc., 2010-03-01)
      Many hypotheses have been raised regarding the pathophysiology of Alzheimer’s disease (AD). Because amyloid beta peptide (Ab) deposition in senile plaques appears as a late, nonspecific event, recent evidence points to tau ...