Browsing by Author "Alvarez Lobos, Manuel"

Now showing items 1-3 of 3

    • Clinical and genetic features of hereditary periodic fever syndromes in Hispanic patients: The Chilean experience 
      Vergara, Cristian; Borzutzky, Arturo; Gutierrez, Miguel; Iacobelli, Sergio; Talesnik, Eduardo; Martínez, María; Stange, Lilith; Basualdo, Javier; Maluje, Viviana; Jimenez, Renato; Wiener, Roberto; Tinoco, Javier; Jarpa, Elena; Aróstegui, Juan; Yagüe, Jordi; Alvarez Lobos, Manuel (2012)
      Hereditary periodic fever syndromes (HPFS) are rare genetic diseases characterized by recurrent episodes of inflammation. Little information is available concerning HPFS in Latin American Hispanic population. The purpose ...
    • Consenso chileno de prevención, diagnóstico y tratamiento de la diarrea asociada a Clostridium difficile 
      Hernandez Rocha, Cristian; Pidal P, Paola; Ajenjo, Cristina; Quera Pino, Rodrigo; Quintanilla, Marcela; Lubascher, Jaime; Jemenao Pacheco, María; Ibáñez, Patricio; Alvarez Lobos, Manuel; Diomedi, Alexis; Marcotti, Alejandra; Acuña Avila, Mirta; Arab, Juan; Riquelme, Arnoldo; Candia, Roberto; Carvajal, Sergio (Sociedad Chilena de Infectología, 2016)
      Clostridium difficile-associated diarrhea (CDAD) has become very important due to the increase in its incidence, severity, recurrence and the associated economic burden. Having a national consensus guideline is essential ...
    • Increased production of soluble TLR2 by lamina propria mononuclear cells from ulcerative colitis patients 
      Candia, Enzo; Díaz Jiménez, David; Langjahr, Patricia; Núñez, Lucía E.; Fuente, Marjorie de la; Farfán, Nancy; López Kostner, Francisco; Abedrapo Moreira, Mario Antonio; Alvarez Lobos, Manuel; Pinedo, George; Beltrán, Caroll J.; González, Carlos; González Burgos, María Julieta; Quera Pino, Rodrigo (2012)
      Toll-like receptor 2 (TLR2) is a type I pattern recognition receptor that has been shown to participate in intestinal homeostasis. Its increased expression in the lamina propria has been associated with the pathogenesis ...