Browsing by Author "Wilson, Carlos"
Now showing items 1-6 of 6
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Wilson, Carlos; Muñoz Palma, Ernesto; Henríquez, Daniel R.; Palmisano, Ilaria; Núñez González, Marco; Di Giovanni, Simone; González Billault, Christian (Soc Neuroscience, 2016)Physiological levels of ROS support neurite outgrowth and axonal specification, but the mechanisms by which ROS are able to shape neurons remain unknown. Ca2+, a broad intracellular second messenger, promotes both Rac1 ...
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Wilson, Carlos; Núñez González, Marco; González Billault, Christian (Company of Biologists Ltd., 2015)Reactive oxygen species (ROS) produced by the NADPH oxidase (NOX) complex play important physiological and pathological roles in neurotransmission and neurodegeneration, respectively. However, the contribution of ROS to ...
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Altamirano, Francisco; Oyarce, César; Silva, Patricia; Toyos, Marcela; Wilson, Carlos; Lavandero González, Sergio; Uhlen, P.; Estrada, M. (BIOSCIENTIFICA LTD, 2009-09)Correction.
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Muñoz Llancao, Pablo; Henríquez, Daniel R.; Wilson, Carlos; Bodaleo, Felipe; Boddeke, Erik W.; Lezoualc'h, Frank; Schmidt, Martina; González Billault, Christian (Soc. Neuroscience, 2015)Acquisition of neuronal polarity is a complex process involving cellular and molecular events. The second messenger cAMP is involved in axonal specification through activation of protein kinase A. However, an alternative ...
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Wilson, Carlos; González Billault, Christian (Frontiers Media, 2015)A proper balance between chemical reduction and oxidation (known as redox balance) is essential for normal cellular physiology. Deregulation in the production of oxidative species leads to DNA damage, lipid peroxidation ...
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Altamirano, Francisco; Oyarce, César; Silva, Patricio; Toyos, Marcela; Wilson, Carlos; Lavandero González, Sergio; Uhlen, L.; Estrada, M. (BIOSCIENTIFICA LTD, 2009-08)Elevated testosterone concentrations induce cardiac hypertrophy but the molecular mechanisms are poorly understood. Anabolic properties of testosterone involve in increase in protein synthesis. The mammalian target of ...