Redox control of brain calcium in health and disease 

NADPH oxidase and hydrogen peroxide mediate insulin-induced calcium increase in skeletal muscle cells 

High-frequency field stimulation of primary neurons enhances ryanodine receptor-mediated Ca2+ release and generates hydrogen peroxide, which jointly stimulate NF-κB activity 

Amyloid β-peptide oligomers stimulate RyR-mediated Ca2+ release inducing mitochondrial fragmentation in hippocampal neurons and prevent RyR-mediated dendritic spine remodeling produced by BDNF 

Crosstalk between calcium and redox signaling: From molecular mechanisms to health implications 

A role for reactive oxygen/nitrogen species and iron on neuronal synaptic plasticity 

Contribution of Ca2+ release channels to hippocampal synaptic plasticity and spatial memory: Potential redox modulation