Browsing by Author "Galluzzi, Lorenzo"

Now showing items 1-6 of 6

    • Erratum to: Mitochondrial control of cell death induced by hyperosmotic stress 
      Criollo Céspedes, Alfredo; Galluzzi, Lorenzo; Chiara Maiuri, M.; Tasdemir, Ezgi; Lavandero González, Sergio; Kroemer, Guido (Springer Science+Business, 2013)
      Errors were inadvertently included in Fig. 6b of the article mentioned above. In particular, the immunoblots depicting the levels of Bcl-XL and Mcl-1, which were originally intended to constitute loading controls for ...
    • The IKK complex contributes to the induction of autophagy 
      Criollo Céspedes, Alfredo; Senovilla, Laura; Authier, Helene; Maiuri, M. C.; Morselli, E.; Vitale, I.; Kepp, O.; Tasdemir, E.; Galluzzi, Lorenzo; Shen, S.; Tailler, M.; Delahaye, N.; Tesniere, A.; De Stefano, D.; Ben Younes, Amena; Harper, F.; Pierron, G.; Lavandero González, Sergio; Zitvogel, L.; Israel, A; Baud, V; Kroemer, Guido (NATURE PUBLISHING GROUP, 2010-02-03)
      In response to stress, cells start transcriptional and transcription-independent programs that can lead to adaptation or death. Here, we show that multiple inducers of autophagy, including nutrient depletion, trigger the ...
    • The inositol 1,4,5-trisphosphate receptor regulates autophagy through its interaction with Beclin 1 
      Vicencio, José Miguel; Ortíz, C.; Criollo Céspedes, Alfredo; Jones, A. W. E.; Kepp, O.; Galluzzi, Lorenzo; Joza, N.; Vitale, I.; Morselli, E.; Tailler, M.; Castedo, M.; Maiuri, M. C.; Molgó, Jordi; Szabadkai, G.; Lavandero González, Sergio; Kroemer, Guido (NATURE PUBLISHING GROUP, 2009-07)
      The inositol 1,4,5-trisphosphate receptor (IP3R) is a major regulator of apoptotic signaling. Through interactions with members of the Bcl-2 family of proteins, it drives calcium (Ca2+) transients from the endoplasmic ...
    • Mitochondrial control of cell death induced by hyperosmotic stress 
      Criollo Céspedes, Alfredo; Galluzzi, Lorenzo; Maiuri, M. C.; Tasdemir, E.; Lavandero González, Sergio; Kroemer, Guido (SPRINGER, 2007-01)
      HeLa and HCT116 cells respond differentially to sorbitol, an osmolyte able to induce hypertonic stress. In these models, sorbitol promoted the phenotypic manifestations of early apoptosis followed by complete loss of ...
    • Senescence, Apoptosis or Autophagy? 
      Vicencio, José Miguel; Galluzzi, Lorenzo; Tajeddine, Nicolás; Ortiz, Carla; Criollo Céspedes, Alfredo; Tasdemir, E.; Morselli, Eugenia; Ben Younes, Amena; Chiara Maiuri, María; Lavandero González, Sergio; Kroemer, Guido (S. Karger AG, 2008)
      Many features of aging result from the incapacity of cells to adapt to stress conditions. When damage accumulates irreversibly, mitotic cells from renewable tissues rely on either of two mechanisms to avoid replication. ...
    • Unsaturated fatty acids induce non-canonical autophagy 
      Niso Santano, Mireia; Malik, Shoaib; Pietrocola, Federico; Bravo San Pedro, José; Mariño, Guillermo; Cianfanelli, Valentina; Ben-Younès, Amena; Troncoso Cotal, Rodrigo; Markaki, Maria; Sica, Valentina; Izzo, Valentina; Chaba, Kariman; Bauvy, Chantal; Dupont, Nicolas; Kepp, Oliver; Rockenfeller, Patrick; Wolinski, Heimo; Madeo, Frank; Lavandero González, Sergio; Codogno, Patrice; Harper, Francis; Pierron, Gérard; Tavernarakis, Nektarios; Cecconi, Francesco; Maiuri, Maria Chiara; Galluzzi, Lorenzo; Kroemer, Guido (Wiley-Blackwell, 2015)
      To obtain mechanistic insights into the cross talk between lipolysis and autophagy, two key metabolic responses to starvation, we screened the autophagy-inducing potential of a panel of fatty acids in human cancer cells. ...