Browsing by Author "Estrada Hormazábal, Manuel"
Now showing items 1-6 of 6
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Americo Da Silva, Luan; Aguilera, Javiera; Quinteros Waltemath, Oscar Alberto; Sánchez Aguilera, Pablo Ignacio; Russell, Javier; Cadagan Fuentes, Cynthia Angelica; Meneses Valdés, Roberto Andrés; Sánchez Vergara, Jeannette Romina; Estrada Hormazábal, Manuel; Jorquera, Gonzalo; Barrientos Briones, Genaro Christian; Llanos Vidal, Paola (MDPI, 2021)Low-grade chronic inflammation plays a pivotal role in the pathogenesis of insulin resistance (IR), and skeletal muscle has a central role in this condition. NLRP3 inflammasome activation pathways promote low-grade chronic ...
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Martínez, Ramón; Scaglione, M.; Luneburg F., Carlos; Hernández A., Eduardo; Araneda V., Oscar; González S.L, Max; Estrada Hormazábal, Manuel; White Ortiz, Allan (Universidad de Chile, 2000)
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Estrada Hormazábal, Manuel; Espinosa, Alejandra; Gibson, C. J.; Uhlen, P.; Jaimovich Pérez, Enrique (SOC ENDOCRINOLOGY, 2005-02)Ca2+ oscillations are one of the most important signals within the cell. The mechanism for generation of Ca2+ oscillations is still not yet fully elucidated. We studied the role of capacitative Ca2+ entry (CCE) on intracellular ...
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Heidrich, Felix M.; Zhang, Kun; Estrada Hormazábal, Manuel; Huang, Yan; Giordano, Frank J.; Ehrlich, Barbara E. (Lippincott Williams & Wilkins, 2008)Altered regulation of signaling pathways can lead to pathologies including cardiac hypertrophy and heart failure. We report that neonatal and adult cardiomyocytes express chromogranin B (CGB), a Ca2+ binding protein that ...
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Durán, Javier; Estrada Hormazábal, Manuel (Frontiers Media, 2018)
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Ibarra, Cristián; Vicencio, José M.; Estrada Hormazábal, Manuel; Lin, Yingbo; Rocco, Paola; Rebellato, Paola; Muñoz, Juan P.; García Prieto, Jaime; Quest, Andrew F. G.; Chiong Lay, Mario; Davidson, Sean M.; Bulatovic, Ivana; Grinnemo, Karl Henrik; Larsson, Olle; Szabadkai, Gyorgy; Uhlén, Per; Jaimovich Pérez, Enrique; Lavandero González, Sergio (American Heart Association, 2013)Rationale: The ability of a cell to independently regulate nuclear and cytosolic Ca2+ signaling is currently attributed to the differential distribution of inositol 1,4,5-trisphosphate receptor channel isoforms in the ...