Browsing by Subject "Endothelium"

Now showing items 1-4 of 4

    • Dexmedetomidine protects the heart against ischemia-reperfusion injury by an endothelial eNOS/NO dependent ... 
      Riquelme, Jaime; Westermeier, Francisco; Hall, Andrew R.; Vicencio, Jose; Pedrozo Cibils, Zully; Ibacache, Mauricio; Fuenzalida, Barbara; Sobrevia, Luis; Davidson, Sean M.; Yellon, Derek M.; Sánchez, Gina; Lavandero González, Sergio (Elsevier, 2016)
      The alpha2-adrenergic receptor agonist Dexmedetomidine (Dex) is a sedative medication used by anesthesiologists. Dex protects the heart against ischemia-reperfusion (IR) and can also act as a preconditioning mimetic. The ...
    • Relative participation of adenosine and endothelium derived mediators in coronary reactive hyperemia in the dog 
      Macho Fisher, Pilar; Domenech Lira, Raúl; Penna Varela, Mario (1995)
      The metabolites that mediate coronary reactive hyperemia have not been definitely identified. Although adenosine and endothelium derived substances seem to be involved, their relative contributions have not been defined ...
    • Subclinical endothelial inflammation markers in a family with type I familial hyperaldosteronism caused by ... 
      Stehr, Carlos B.; Carvajal, Cristián A.; Lacourt, Patricia; Alcaíno Olivares, Hernán Alejandro; Mellado, Rose Marie; Cattani, Andreína; Mosso, Lorena; Lavandero González, Sergio; Fardella, Carlos E. (SOC MEDICA SANTIAGO, 2008-09)
      Background: Type I familial hyperaldosteronism is caused by the presence of a chimaeric gene CYP11B1/CYP11B2 which encodes an enzyme with aldosterone synthetase activity regulated by adernocorticotrophic hormone (ACTH). ...
    • Targeting the Endothelium to Achieve Cardioprotection 
      Herrera Zelada, Nicolás Gabriel; Zúñiga Cuevas, Úrsula Almendra; Ramírez Reyes, Andrés Ignacio; Lavandero González, Sergio; Riquelme Meléndez, Jaime (Frontiers Media, 2021)
      Despite considerable improvements in the treatment of myocardial infarction, it is still a highly prevalent disease worldwide. Novel therapeutic strategies to limit infarct size are required to protect myocardial function ...