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Authordc.contributor.authorOcaranza, María Paz 
Authordc.contributor.authorRivera, Paulina es_CL
Authordc.contributor.authorNovoa, Ulises es_CL
Authordc.contributor.authorPinto, Melissa es_CL
Authordc.contributor.authorGonzález, Leticia es_CL
Authordc.contributor.authorChiong Lay, Mario es_CL
Authordc.contributor.authorLavandero González, Sergioes_CL
Authordc.contributor.authorJalil Milad, Jorge es_CL
Admission datedc.date.accessioned2011-08-08T13:34:12Z
Available datedc.date.available2011-08-08T13:34:12Z
Publication datedc.date.issued2011-04
Cita de ítemdc.identifier.citationJOURNAL OF HYPERTENSION 29 (4): 706-715es_CL
Identifierdc.identifier.issn0263-6352
Identifierdc.identifier.urihttp://repositorio.uchile.cl/handle/2250/121576
General notedc.descriptionArtículo de publicación ISIes_CL
Abstractdc.description.abstractBackground Angiotensin II (Ang II) levels depend on renin, angiotensin-converting enzyme (ACE), and on the homologous angiotensin-converting enzyme (ACE2). Increased ACE and Ang II levels are associated with higher Rho kinase activity. However, the relationship between Rho kinase activation and ACE2 in hypertension is unknown. Objective The role of the Rho kinase signaling pathway in both enzymatic activity and aortic gene expression of ACE2 in deoxycorticosterone acetate (DOCA) hypertensive rats was assessed in the present study.es_CL
Lenguagedc.language.isoenes_CL
Publisherdc.publisherLIPPINCOTT WILLIAMS & WILKINSes_CL
Keywordsdc.subjectPULMONARY ARTERIAL-HYPERTENSIONes_CL
Títulodc.titleRho kinase inhibition activates the homologous angiotensin-converting enzyme-angiotensin-(1-9) axis in experimental hypertensiones_CL
Document typedc.typeArtículo de revistaes_CL


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