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Authordc.contributor.authorCarpentier, 
Authordc.contributor.authorNarvarte, 
Authordc.contributor.authorSanhueza, 
Admission datedc.date.accessioned2019-03-18T12:02:16Z
Available datedc.date.available2019-03-18T12:02:16Z
Publication datedc.date.issued1977
Cita de ítemdc.identifier.citationCardiovascular Research, Volumen 11, Issue 1, 2018, Pages 39-46
Identifierdc.identifier.issn00086363
Identifierdc.identifier.other10.1093/cvr/11.1.39
Identifierdc.identifier.urihttps://repositorio.uchile.cl/handle/2250/167473
Abstractdc.description.abstractSummary: The mechanism of the transient enhancement of the amplitude of the action potential (AAP) induced by harmaline (HME) was studied in rat atria. The results show that HME increases AAP through an enhancement of the slow component responsible for the last part of the upstroke, which overcomes an inhibitory action on the initial fast component. The stimulatory effect on the slow component is mediated through adrenergic beta receptors and normally masks an alpha-dependent depressant effect.
Lenguagedc.language.isoen
Publisherdc.publisherOxford University Press
Type of licensedc.rightsAttribution-NonCommercial-NoDerivs 3.0 Chile
Link to Licensedc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
Sourcedc.sourceCardiovascular Research
Keywordsdc.subjectPhysiology
Keywordsdc.subjectCardiology and Cardiovascular Medicine
Keywordsdc.subjectPhysiology (medical)
Títulodc.titleCatechol-mediated effects of harmaline on the action potential of rat atrial fibres
Document typedc.typeArtículo de revista
Catalogueruchile.catalogadorSCOPUS
Indexationuchile.indexArtículo de publicación SCOPUS
uchile.cosechauchile.cosechaSI


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Attribution-NonCommercial-NoDerivs 3.0 Chile
Except where otherwise noted, this item's license is described as Attribution-NonCommercial-NoDerivs 3.0 Chile