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Authordc.contributor.authorSmith Schuster, Patricio C. 
Authordc.contributor.authorSantibañez, Juan F. es_CL
Authordc.contributor.authorMorales, Juan P. es_CL
Authordc.contributor.authorMartínez, Jorge es_CL
Admission datedc.date.accessioned2014-01-10T13:49:47Z
Available datedc.date.available2014-01-10T13:49:47Z
Publication datedc.date.issued2004-04-08
Cita de ítemdc.identifier.citationJ Periodont Res 2004; 39; 380–387en_US
Identifierdc.identifier.otherdoi: 10.1111/j.1600-0765.2004.00753.x
Identifierdc.identifier.urihttps://repositorio.uchile.cl/handle/2250/123507
General notedc.descriptionArtículo de publicación ISI.en_US
Abstractdc.description.abstractRegulation of the extracellular matrix turnover is a crucial process in wound healing and the progress of periodontal disease. It has been proposed that urokinase-type plasminogen activator (uPA), under the control of growth factors or cytokines, provides the proteolytic potential to the accomplishment of these cellular events. Epidermal growth factor (EGF) is one of the growth factors that has been shown to be active in uPA regulation.en_US
Lenguagedc.language.isoenen_US
Publisherdc.publisherBlackwell Munksgaard Ltden_US
Type of licensedc.rightsAttribution-NonCommercial-NoDerivs 3.0 Chile*
Link to Licensedc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/*
Keywordsdc.subjectc-Jun N-terminal kinase; curcumin; epidermal growth factor; genistein; gingival fibroblasts; urokinaseen_US
Títulodc.titleEpidermal growth factor stimulates urokinase-type plasminogen activator expression in human gingival fibroblasts. Possible modulation by genistein and curcuminen_US
Document typedc.typeArtículo de revista


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Attribution-NonCommercial-NoDerivs 3.0 Chile
Except where otherwise noted, this item's license is described as Attribution-NonCommercial-NoDerivs 3.0 Chile