Browsing by Author "7a7d76cc-490f-44d8-84b6-477a539c7a75"

Now showing items 1-4 of 4

    • Characterization of 14-3-3 isoforms expressed in the Echinococcus granulosus pathogenic larval stage 
      Teichmann, Aline; Vargas, Daiani M.; Monteiro, Karina M.; Meneghetti, Bruna V.; Dutra, Cristine S.; Paredes, Rodolfo; Galanti Garrone, Norbel; Zaha, Arnaldo; Ferreira, Henrique B. (American Chemical Society, 2015)
      © 2015 American Chemical Society.The 14-3-3 protein family of eukaryotic regulators was studied in Echinococcus granulosus, the causative agent of cystic hydatid disease. These proteins mediate important cellular processes ...
    • In vitro segmentation induction of Mesocestoides corti (cestoda) tetrathyridia 
      Markoski, Melissa M.; Bizarro, Cristiano V.; Farias, Sandra; Espinoza, Ingrid; Galanti Garrone, Norbel; Zaha, Arnaldo; Ferreira, Henrique B. (2003)
      Mesocestoides corti is a suitable model for studying cestode development because of its ability to reproduce asexually and segment in vitro. The cultured parasite is also capable of sexual differentiation and, probably, ...
    • New insights of the local immune response against both fertile and infertile hydatid cysts 
      Hidalgo, Christian; Stoore, Caroll; Strull, Karen; Franco, Carmen; Corrêa, Felipe; Jiménez, Mauricio; Hernández, Marcela; Lorenzatto, Karina; Ferreira, Henrique B.; Galanti Garrone, Norbel; Paredes, Rodolfo (Public Library of Science, 2019)
      Background Cystic echinococcosis is caused by the metacestode of the zoonotic flatworm Echinococcus granulosus. Within the viscera of the intermediate host, the metacestode grows as a unilocular cyst known as hydatid cyst. ...
    • Praziquantel and albendazole damaging action on in vitro developing Mesocestoides corti (Platyhelminthes: Cestoda) 
      Markoski, Melissa M.; Trindade, Edvaldo S.; Cabrera, Gonzalo; Laschuk, Alice; Galanti Garrone, Norbel; Zaha, Arnaldo; Nader, Helena B.; Ferreira, Henrique B. (2006)
      Parasitic flatworms present several steps of body architecture rearrangement during their fast transition from one developmental stage to another, which are, at least in part, responsible for their evasion from host immune ...