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Authordc.contributor.authorRuz, Manuel 
Authordc.contributor.authorCarrasco, Fernando 
Authordc.contributor.authorRojas, Pamela 
Authordc.contributor.authorBasfi-fer, Karen 
Authordc.contributor.authorHernández, Maria Catalina 
Authordc.contributor.authorPérez, Alvaro 
Cita de ítemdc.identifier.citationBiological Trace Element Research, Volumen 188, Issue 1, 2019, Pages 177-188
Abstractdc.description.abstract© 2019, Springer Science+Business Media, LLC, part of Springer Nature.Zinc (Zn) plays crucial roles in mammalian metabolism. There is increasing interest about the potential beneficial effects of Zn on the prevention or treatment of non-communicable diseases. This review critically analyzes the information related to the role of Zn on the metabolic syndrome (MetS) as well as type 2 diabetes (T2D), and summarizes the biological basis of these potential effects of Zn. There are several mechanisms by which Zn may help to prevent the development or progression of MetS and T2D, respectively. Zn is involved in both insulin secretion and action in peripheral tissues. Specifically, Zn has insulin-mimetic properties that increase the activity of the insulin signaling pathway. Zn modulates long-chain polyunsaturated fatty acids levels through its action on the absorption of essential fatty acids in the intestine and its subsequent desaturation. Zn is also involved in both the assembly of chylomi
Publisherdc.publisherHumana Press Inc.
Type of licensedc.rightsAttribution-NonCommercial-NoDerivs 3.0 Chile
Link to Licensedc.rights.uri
Sourcedc.sourceBiological Trace Element Research
Keywordsdc.subjectMetabolic syndrome
Títulodc.titleNutritional Effects of Zinc on Metabolic Syndrome and Type 2 Diabetes: Mechanisms and Main Findings in Human Studies
Document typedc.typeArtículo de revista
Indexationuchile.indexArtículo de publicación SCOPUS

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Attribution-NonCommercial-NoDerivs 3.0 Chile
Except where otherwise noted, this item's license is described as Attribution-NonCommercial-NoDerivs 3.0 Chile